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10X Genomics grcm39 mouse reference genome
Grcm39 Mouse Reference Genome, supplied by 10X Genomics, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/grcm39+mouse+reference+genome/genome+human+reference/bio_rxiv__2025__07__11__664370-56-28-32
Average 86 stars, based on 1 article reviews
grcm39 mouse reference genome - by Bioz Stars, 2026-10
86/100 stars

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Article Title: Single-cell transcriptomics reveal alveolar macrophages-specific responses in single-hit ozone exposure model in mice
Article Snippet: Raw FASTQ files were demultiplexed and the unique transcripts with unique molecular identifiers (UMI) were counted using the Cell Ranger pipeline (v8.0.0), and individual reads were aligned to GRCm39 mouse reference genome (10X genomics, Pleasanton, CA).

Article Title: Induction of a mismatch repair deficient genotype by tailored chemical mutagenesis in experimental models of cancer.
Article Snippet: Article Induction of a mismatch repair deficient genotype by tailored chemical mutagenesis in experimental models of cancer Graphical abstract Highlights • Tailored mutagenic chemical treatment leads to epigenetic loss of Msh2 • Temozolomide plus cisplatin induces high TMB and microsatellite instability • Chemically induced MMRd genotype generates high immunogenicity in preclinical models • In treated patients, MMRd signatures emerged but no responses were observed Authors Benoit Rousseau, Mitesh Patel, Oliver Artz, ..., Elisa de Stanchina, Neil H. Segal, Luis A. Diaz, Jr.. Correspondence diazl5@mskcc.org In brief Rousseau et al. show that tailored mutagenic chemical treatment induces a mismatch repair deficient genotype by epigenetic Msh2 silencing leading to potent immune response in immunoresistant tumor models and sensitizing to PD-1 blockade.. Rousseau et al., 2025, Cancer Cell 43, 1–15 July 14, 2025 © 2025 Elsevier Inc. All rights are reserved, including those for text and data mining, AI training, and similar technologies. https://doi.org/10.1016/j.ccell.2025.05.010 ll



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